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Peripheral neuroimmune alterations associated with cognitive vulnerability in Long COVID
Vol. 1 2024 -347365 -
Acute IFN-γ responses predict and mediate chronic pain caused by arthritogenic alphavirus infection
-346499 -
Role of TLR4 polymorphisms and gut–brain axis signaling in the therapeutic response to natalizumab in multiple sclerosis
-346164 -
SARS-CoV-2 Spike Protein Induces an EAE-Like Model of Persistent Pain Through Neuroinflammation and Synaptic Remodeling
-343913
Publications in other proceedings
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GeneReg 2025
GENETIC POLYMORPHISMS IN THE VDR GENE AND THEIR INFLUENCE ON NATALIZUMAB RESPONSE IN MULTIPLE SCLEROSIS PATIENTS
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Inflammation
Unraveling the Mediators of Persistent Pain Induced by CHIKV: The Role of IFNγ in Human and Experimental Models
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BCTRIMS 2024
FCGR2A rs1801274 polymorphism is associated with response to natalizumab in multiple sclerosis patients
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FRONTIERS 2024
Genetic variants in FCGR2A, PTPN2, VDR as Predictive Signatures of Aggressive Phenotypes in Cerebral Cavernous Malformation
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FRONTIERS 2024
FCGR2A variant rs1801274 as a Pharmacogenetic Predictor of Natalizumab Therapeutic Failure in Multiple Sclerosis Patients
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